Herpes simplex
☆ AI Dermatology — Free Service
Muzotsatira za 2022 Stiftung Warentest zochokera ku Germany, kukhutitsidwa kwa ogula ndi ModelDerm kunali kotsika pang'ono kusiyana ndi kuyankhulana kwa telemedicine komwe kulipiridwa.
Muzotsatira za 2022 Stiftung Warentest zochokera ku Germany, kukhutitsidwa kwa ogula ndi ModelDerm kunali kotsika pang'ono kusiyana ndi kuyankhulana kwa telemedicine komwe kulipiridwa.
Herpes simplex pa milomo.
Herpes simplex ― Matenda a Herpes simplex pa zala amapezeka kwambiri m'ana a m'maiko kuposa akuluakulu.
relevance score : -100.0%
ReferencesMatenda a HSV-1 amapita patsogolo kuchokera pa zovuta za epithelial, kenako amawonjezera latency, makamaka mu neurons, ndipo amafika mu kubwezeretsanso. HSV-1 nthawi zambiri imayambitsa kuphuka koyamba ndi kuphuka kwa vesicular, makamaka pa mphuno ndi mucosa ya m'mutu. Zochitika zake zimachokera ku orolabial herpes ndipo zimatha kuyambika m'magulu osiyanasiyana monga herpetic folliculitis, matenda a pakhungu, kuthekera kwa maso, komanso milandu yovuta ngati nsungu encephalitis. Chithandizo cha antiviral chimathandiza kuthana ndi matenda a HSV.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 2 (HSV-2) ndi matenda ofala, okhudza pafupifupi 22% ya akuluakulu a zaka 12 ndi kupitilira, okwana 45 miliyoni akulu ku United States. Ngakhale kuti HSV-1 nthawi zambiri imayambitsa zilonda za'kamwa, imatha kuyambitsa zilonda za'mimba. Komabe, pamene odwala ali ndi zotupa kumaliseche, HSV-2 nthawi zambiri imakhala yodetsa nkhawa kwambiri. Zizindikiro za kuphulika kwa HSV-2 nthawi zambiri zimakhala zosamveka bwino, monga kuyabwa ndi kukwiya kwa maliseche, zomwe zingachedwe kuzindikira ndi kulandira chithandizo. Kuchedwa kumeneku kungayambitse kufalikira kwa anthu omwe alibe kachilomboka.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus (HSV) nthawi zambiri imayambitsa matenda monga maliseche ndi zilonda zozizira kwa achinyamata ndi akulu. Pamene HSV imayamba pa khanda mkati mwa masabata 4‑6 a moyo, imatha kudwala kwambiri ndi zotsatira zake zoopsa. Kuzindikira nthawi ya matenda a HSV pa khanda ndi kofunika kuti matendawa asapitirire, kuteteza matenda omwe amatha kumaliza (ngakhale imfa).
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex ndi matenda omwe amapezeka kwambiri ndipo amatha kulalikira kuchokera kwa amayi omwe ali ndi chizindikiro cha matenda kupita kwa ana awo. Kachilombo kamatha kuyambitsa matenda aakulu kapena imfa mwa ana omwe ali pa chiwopsezo. Ngakhale kuti sizochitika nthawi zonse, nthawi zambiri zimachitika panthawi yotulutsa. Chiwopsezo chimakhala chachikulu ngati mayi atenga kachilombo panthawi yomwe akukula. Komabe, chiopsezo ichi chitha kuchepetsa pogwiritsa ntchito mankhwala oletsa tizilombo toyambitsa matenda kapena kusankha gawo la C nthawi zina.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Herpes simplex virus (HSV) mitundu 1 ndi 2 imakhudza anthu ambiri padziko lonse lapansi. Nthawi zambiri, kachilomboka kamakhala chete m'maselo a mitsempha pambuyo pa kupirira pakhungu, koma imatha kuyambiranso pambuyo pake, kumayambitsa zilonda zozizira. Nthawi zina, zimabweretsa mavuto akulu monga matenda a maso, kutupa mu ubongo, kapena mikhalidwe yowopsa kwa ana odwala komanso anthu omwe ali ndi chitetezo chochuluka. Ngakhale mankhwala omwe alipo amathandizira kuwongolera matenda, chiwopsezo cha kukana mankhwala ndi zotsatira zake chimakhalabe chofunika. Tikufuna mankhwala atsopano kuti tithane ndi kachilomboka bwino.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Pali mitundu iwiri ya kachilombo ka herpes simplex, mtundu 1 (HSV-1) ndi mtundu wachiwiri (HSV-2). HSV-1 nthawi zambiri imayambitsa matenda pakamwa, pomwe HSV-2 imayambitsa matenda kumaliseche. Zimafalitsidwa mwachindunji ndi munthu amene ali ndi kachilombo. Genital herpes imasonyeza ngati matenda opatsirana pogonana. Akhoza kufalikira kwa khanda panthawi yobereka. Pambuyo pa matenda, ma virus amasamutsidwa m'mitsempha ya minyewa kupita ku ma cell a mitsempha, komwe amakhala moyo wonse. Zinthu zomwe zimayambitsa kubwereza zingaphatikizepo: kuchepa kwa chitetezo cha thupi, kupsinjika maganizo, ndi kuwala kwa dzuwa.
Nthawi zambiri, mankhwala oletsa tizilombo toyambitsa matenda amatengedwa pokhapokha ngati zizindikiro zakhala zovuta kwambiri. Mankhwala oletsa tizilombo toyambitsa matenda tsiku ndi tsiku akhoza kuperekedwa kwa munthu amene ali ndi matenda afupifupi. Palibe katemera wopezeka, ndipo katemera wa shingles samateteze herpes simplex. Kuchiza ndi mankhwala oletsa tizilombo toyambitsa matenda monga aciclovir kapena valaciclovir kumatha kuchepetsa kuopsa kwa zizindikiro.
Padziko lonse lapansi, HSV-1 kapena HSV-2 ali pakati pa 60 % ndi 95 % a akulu. HSV-1 nthawi zambiri amadwala pa ana. Pafupifupi anthu 536 millioni padziko lonse lapansi (16 % ya anthu) adadwala HSV-2 pa chaka cha 2003, ndipo chiwopsezo chachikulu chili pakati pa amayi ndi anthu okula. Anthu ambiri omwe ali ndi HSV-2 samazindikira kuti ali ndi kachilombo.
○ Chithandizo ― OTC Mankhwala
Pewani kulumikizana kulikonse, monga kupsompsona mwana, pamene matuza alipo, chifukwa kulumikizana kungathe kufalitsa matenda kwa anthu ena. Muyenera kupuma osamwa mowa.
#Acyclovir cream